Buffers intramuscular acidosis; chelates divalent metals; quenches reactive carbonyls (anti-glycation).
Reference facts
- Also known as
- β-Alanyl-L-histidine
- Category
- Neuropeptide & Other
- Mechanism of action
- Buffers intramuscular acidosis; chelates divalent metals; quenches reactive carbonyls (anti-glycation).
- Targets
- Multiple — non-receptor
- Indications under research or approved
- Research — exercise performance (β-alanine precursor), longevity, diabetic complications
- Regulatory status (reference entry)
- Dietary supplement; not FDA-approved as drug
- Half-life
- Minutes (intact); rapidly hydrolyzed by serum carnosinase
- Sequence notes
- Dipeptide β-Ala-His. β-Alanine is non-α-amino, so single-letter code does not apply.
- Molecular weight (Da)
- 226
- First reported
- 1900
Primary References
Explore research articles about Carnosine
This is an educational reference, not medical advice. Research findings do not establish safety or efficacy for self-administration. Consult the primary sources for study populations, limitations, and current regulatory status.